A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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Thromb Haemost, 71pp. The Kinston antiphospholipid group. Conclusions Epidemiological and laboratory enffermedad from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint. Antithrombin heparin cofactor assay with new chromogenic substrates.
It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis.
J Lab Clin Med,pp.
To improve our services and products, we use “cookies” own or third parties authorized to show advertising related to client preferences through the analyses of navigation customer behavior. Association of idiopathic venous thromboembolism with single point-mutation at Arg of factor. Thromb Haemost, 78pp. J Med,pp. Aguirre Canyadell aI. Br J Hematol, 71pp. J Biol Chem,pp. High risk of thrombosis in patients homozigous for factor V Leiden activated protein C resistance.
Mutation in blood coagulation factor V associated with resistance to activated protein C. Maturation of perthss hemostatic system during childhood. Thromb Res, 11pp. Mutation in gene coding for factor V and the risk of myocardial infarction, stroke and venous thrombosis in apparently healthy men.
J Clin Invest, 94pp. Clin Othop,pp. Blood, 87pp. Special laboratory evaluation of coagulation. J Pediatr Orthop, 19pp. Non traumatic osteonecrosis of the femoral head: Blood collection in strongly acid citrate anticoagulant used in a study of dietary influence of basal Tpa activity. Acta Med Scand,pp. Background It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a efermedad hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis.
Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Anal Biochem,pp.
Perthes’ disease and the relevance of thrombophilia. N Engl J Med,pp. Genetics aspects of Perthes disease: The remaining patients were considered withinthe normal range when age was taken into account.
Are you a health professional able to prescribe or dispense drugs? Blood, 82pp. Clin Orthop,pp. Association of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease. Activated protein C resistance as an additional risk factor for thrombosis in protein C deficient families. Lancet,pp.
Síndrome de Legg-Calvé-Perthes – Wikipédia, a enciclopédia livre
Determination of plasminogen activator and its fast inhibitor in plasma. Thromb Haemost, 62pp. Am J Hematol, 45pp.
Nature,pp. You can change the settings or obtain more information by clicking here. Patients and method Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients. Thromb Haemost, 69pp.
Relation of altered hemostasis to etiology.
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Pediatr Res, 35pp. Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients. Blood, 85pp. Am J Hematol, 44pp.